Crohn's Disease (CD) and Ulcerative Colitis (UC), are both forms of Inflammatory bowel disease (IBD) that disrupt the body's ability to digest food, absorb nutrients, and eliminate waste in a healthy manner. The causes of CD and UC are complex but include a combination of environmental, immune and bacterial factors in genetically susceptible individuals. An estimated 1.4 million people suffer from IBD in the United States alone. Symptoms include diarrhea, abdominal pains and cramping, and intestinal ulcers.

BLOG: https://www.ergopathics.com/blogs/news/crohns-disease-colitis-new-kit

KIT CONTENTS

Adaptive Immunity

Autoimmunity

“Autoinflammatory diseases are dysfunctions of the ‘generalized immune system.’ While an autoimmune disease occurs when the immune system attacks healthy cells, autoinflammatory diseases involve an apparent antigen-independent hyperactivation of the innate immune system. The innate immune system is the body’s first line of defence against infection. When microbes invade the body, the innate immune system quickly responds by triggering fever and inflammation, which help the body fight infection.” https://www.wehi.edu.au/research-diseases/immune-health-and-infection/autoinflammatory-diseases

Bacteria – Bacteroidales

Bacteria - Clostridia

Bacteria – Enterobacteriacea

Some potentially harmful microbial species were more abundant in Crohn's patients, such as those belonging to the Enterobacteriaceae, Pasteurellaceae, Veillonellaceae, and Fusobacteriaceae; numbers of the Erysipelotrichales, Bacteroidales, and Clostridiales, generally considered to be beneficial, were lower.  https://www.science.org/content/article/crohns-disease-marked-dramatic-changes-gut-bacteria

Bacteria – Erysipelotrichales

Bacteria – Fusobacteriaceae

Bacteria – Pasteurellaceae

Bacteria – Veillonellaceae

Bifidobacterium Mix

Bacteria – Adherent-Invasive Escherichia coli (AIEC)

“Adherent-invasive E. coli colonize the intestinal mucosa by adhering to intestinal epithelial cells. They are also true invasive pathogens, able to invade intestinal epithelial cells and to replicate intracellularly.” https://pubmed.ncbi.nlm.nih.gov/17133079/#

Bacteria - Faecalibacterium prausnitzii

Bacteria - Mycobacterium avium

Bacteria - Mycobacterium Paratuberculosis (MAP)

Studies have suggested a causal role for Mycobacterium avium subspecies paratuberculosis (MAP) which causes a similar disease, Johne’s disease, in cattle. In many individuals, genetic factors predispose individuals this infection.  

Bacteria - Microbial Manna

Biofilms (LWP) Master Vial

Biofilm - Escherichia Coli

Celiac Disease Nosode

 

Colitis, Ulcerative gene – IL10

Ulcerative colitis is a chronic inflammatory bowel disease (IBD) in which abnormal reactions of the immune system cause inflammation and ulcers on the inner lining of your large intestine. Ulcerative colitis can develop at any age, but the disease is more likely to develop in people between the ages of 15 and 30.

https://www.immunology.org/public-information/bitesized-immunology/immune-dysfunction/inflammatory-bowel-disease

Colitis, Ulcerative gene – ARPC2

Colitis, Ulcerative gene – ECM1

Colitis, Microscopic

Colitis, Collagenous

a form of Microscopic Colitis

Colitis, Lymphocytic

 a form of Microscopic Colitis

Colitis, Indeterminate

Colitis, Infectious

A subtype of infectious colitis is Clostridioides difficile colitis, which is informally abbreviated as "C-diff colitis". It classically forms pseudomembranes and is often referred to as pseudomembranous colitis.

Colitis, Ischemic

Related to a lack of blood flow.

Colitis, Pseudomembranous

Related to Clostridium difficile infection (now called Clostridiodes difficile)

Crohn's Disease, Ileocecal

Reported to account for fifty percent of Crohn’s cases. Crohn's ileitis, manifest in the ileum only, accounts for about thirty percent of cases.

Crohn’s Disease gene – NOD2

https://www.immunology.org/public-information/bitesized-immunology/immune-dysfunction/inflammatory-bowel-disease

Crohn’s Disease gene – NLRP3

Crohn’s Disease gene – ATG16L1

In 2007, the ATG16L1 gene was implicated in Crohn's disease, which may induce “autophagy” which may hinder the body's immune system defenses. https://www.gastrojournal.org/article/S0016-5085(07)00558-6/fulltext

Crohn’s Disease gene – IRGM

Protein 1 (IFI1) is an enzyme that in humans is encoded by the IRGM gene 

Crohn’s Disease gene – CCR6

Crohn’s Disease gene – ICOSL

Crohn’s Disease gene – PTGER4

Crohn’s Disease gene – ITLN1

Crohn’s / Colitis gene – IL23R

Crohn’s / Colitis gene – IL12B

Crohn’s / Colitis gene – JAK2

Crohn’s / Colitis gene – STAT6

Crohn’s / Colitis gene – TLR4

Crohn’s / Colitis gene – CARD9

Crohn’s / Colitis gene – IRF5

Crohn’s / Colitis gene – PTPN2/22

Crohn’s / Colitis gene –TNFAIP3

Crohn’s / Colitis gene – IL18RAP

Crohn’s / Colitis gene – LRRK2

Crohn’s / Colitis gene – TNFSF15

Crohn’s / Colitis gene – TNFRSF6B

Cytokine Storm

Dietary, Fiber

Dietary factors can have a significant impact on the gut microbiome. Some studies suggest that a Western diet, a diet higher in refined sugars, processed foods, food additives and lower in dietary fiber, changes the microbiome of our gut. https://crohnsandcolitis.ca/About-Crohn-s-Colitis/Are-you-at-risk

Dietary, Refined Sugar

Drugs, Antibiotics (ANT) Master Vial

In one large retrospective study, patients who were prescribed doxycycline for acne had a 2.25-fold greater risk of developing Crohn's disease

Drugs, Nonsteroidal anti-inflammatory drugs 

Enteric Nervous System

Fecal Matter

Fistulous Tracts

Intraepithelial Lymphocytes 

https://news.cornell.edu/stories/2011/02/common-parasite-uncovers-key-cause-crohns#

Gastroenteritis Nosode

Gastroenteritis, also referred to as "stomach flu, " is inflammation of the stomach and intestinal lining, which causes diarrhea and vomiting. It can be caused by a virus, a bacteria or a parasite.

Granuloma

A granuloma is an aggregation of macrophages that forms in response to chronic inflammation. They are found in those with Crohn’s Disease. This occurs when the immune system attempts to isolate foreign substances that it is otherwise unable to eliminate. https://en.wikipedia.org/wiki/Granuloma

Innate Immunity

https://www.uptodate.com/contents/the-autoinflammatory-diseases-an-overview

Intestine, Microbiome Bacteria

Intestine, Microbiome Fungi

Intestine, Microbiome Protozoa

Intestine, Microbiome Virus

Intestine, Large

Main sections of the LI are: cecum and appendix; ascending colon; hepatic flexure; transverse colon; splenic flexure; descending colon; sigmoid colon; rectum; anus. https://crohnsandcolitis.ca/About-Crohn-s-Colitis/What-are-Crohns-and-Colitis

Intestine, Small

Three major sections of the SI are: duodenum (about 8 cm in length); jejunum (around 3 metres long); and ileum (about 3 metres in length).

Mesentery

Peristalsis

Peristalsis is the involuntary contraction and relaxation of longitudinal and circular muscles throughout the digestive tract, allowing for the propulsion of contents beginning in the pharynx and ending in the anus.

Parasite - Parietal Peritoneum

Parasite - Visceral Peritoneum

Scar, Interference Field

Sympathetic Ganglia

Vagus Nerve

 

 

Quantity: 75 Vials

Model : CDC

Note: Ergopathics test kits are not intended to diagnose, prevent, treat or cure any disease, disorder or abnormal physical state.  The vials contain ethanol and water and the process used to imprint them with electromagnetic signatures has not been tested or validated by any scientific method and is not approved by any regulatory authority.  They are intended for use solely by qualified providers to support wellness and manage stress.